Jason H.T. Bates, Ph.D., D.Sc.

Professor, Department of Medicine, Department of Molecular Physiology & Biophysics; Interim Director, School of Engineering

Room 228, HSRF
University of Vermont, College of Medicine
149 Beaumont Ave.
Burlington, VT. 05405

Voice: (802) 656-8912
Fax: (802) 656-8926

Email: jason.h.bates@uvm.edu

• B.Sc. (honours) (1978) Physics; University of Canterbury, Christchurch, New Zealand.
• Ph.D. (1981) Medicine; Christchurch Clinical School of Medicine, Otago University, Christchurch, New Zealand.
• Post-doc. (1981-1983) Dept Medicine; Christchurch Clinical School of Medicine, Christchurch, New Zealand.
• Post-doc. (1983-1986) Lung Physiology; McGill University Montreal, Quebec, Canada.
• Assistant Professor (1986-1991); Depts of Medicine and Biomedical Engineering, McGill University, Montreal, Quebec, Canada.
• Associate Professor (1991-1999); Depts of Medicine and Biomedical Engineering, McGill University, Montreal, Quebec, Canada.
• Professor (1999); Depts of Medicine and Biomedical Engineering, McGill University, Montreal, Quebec, Canada.
• Research Professor (1999-present); Dept. of Medicine, Dept. of Molecular Physiology & Biophysics, University of Vermont.
• Professor (2007-Present); Dept. Medicine University of Vermont
• Member of Pulmonary/Critical Care Division (1999–present); Fletcher Allen Health Care and University of Vermont Dept. of Medicine.
• Interim Director (2010-Present) School of Engineering University of Vermont

My main research interests focus on lung mechanics and its quantitative evaluation through the application of quantitative methods from physics, engineering and mathematics.

• Lung Mechanics
• Asthma and lung injury
• Mathematical/computer modeling of physiology
• Laboratory instrumentation and measurement

• Acid aspiration-induced airways hyperresponsiveness in mice.
   
   
• Animal models of asthma
   
   
• Transient oscillatory force-length behavior of activated airway smooth muscle
   
   
• The role of time and pressure on alveolar recruitment
   
   
• Neither fibrin nor plasminogen activator inhibitor-1 deficiency protects lung function in a mouse model of acute lung injury
   
   
• Modeling the dynamics of recruitment and derecruitment in mice with acute lung injury
   
   
• A further analysis of why pulmonary venous pressure rises after the onset of LV dysfunction
   
   
• Assessment of peripheral lung mechanics
   
   
• Extracellular matrix mechanics in lung parenchymal diseases
   
   
• Computational assessment of airway wall stiffness in vivo in allergically inflamed mouse models of asthma
   
   
• In silico modeling of interstitial lung mechanics: implications for disease development and repair
   
   
• The synergistic interactions of allergic lung inflammation and intratracheal cationic protein
   
   
• Unrestrained video-assisted plethysmography: a noninvasive method for assessment of lung mechanical function in small animals
   
   
• Heterogeneity of bronchoconstriction does not distinguish mild asthmatic subjects from healthy controls when supine