Robert B. Low, Ph.D.

Dept. Molecular Physiology & Biophysics.

Room: D-207, Given
University of Vermont, College of Medicine
Burlington, VT. 05405

Voice: (802) 656-4338
Fax: (802) 656-0747

Email:Bob.Low@uvm.edu

• A.B. (1963) Biology; Princeton University, Princeton, NJ
• Ph.D. (1968) Physiology; University of Chicago, Chicago, IL
• Postdoc. (1968-70) Biology; Mass. Inst. of Technol., Cambridge, MA
• Assistant Professor (1970-1974); University of Vermont, Dept. of Physiology & Biophysics.
• Associate Professor (1974-1979); University of Vermont, Dept. of Physiology & Biophysics.
• Professor (1979 - present); University of Vermont, Dept. of Molecular Physiology & Biophysics.
• Director, Vermont Pulmonary Specialized Center of Research (SCOR) 1976 - 1992.
• Senior International Fogarty Fellow, 1979 - 1980.
• Research Fellow with Dr. Giulio Gabbiani, Dept. of Pathology, University of Geneva, Geneva, Switzerland, July 1979 - June 1980.
• Associate Dean for Research, College of Medicine, 1984 - 1992.
• Interim Executive Dean, College of Medicine, 1991 - 1992.
• Interim Provost, University of Vermont, October 1992 - June, 1994.
• Provost, University of Vermont, June, 1994 - June, 1996.
• Senior International Fogarty Fellow, 1997 - 1998.
• Research Fellow with Dr. Giulio Gabbiani, Dept. of Pathology, University of Geneva, Geneva, Switzerland, July 1997 - June 1998.

• Smooth muscle cell differentiation
• Function of smooth muscle myosin heavy chain isoforms
• Myosin heavy chain function in cardiomyopathy.

• Microvessel precursor smooth muscle cells express head-inserted smooth muscle myosin heavy chain (SM-B) isoform in hyperoxic pulmonary hypertension.
   
   
• Myofibroblasts in diffuse alveolar damage of the lung.
   
   
• Modulation of myofibroblast and smooth-muscle phenotypes in the lung.
   
   
• Age dependence of smooth muscle myosin expression by cultured rat aortic smooth muscle cells.
   
   
• Regulation of alpha-smooth muscle actin and CRBP-1 expression by retinoic acid and TGF-beta in cultured fibroblasts.
   
   
• Loss of SM-B myosin affects muscle shortening velocity and maximal force development.